AKI and Hyperkalemia
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A client with Acute Kidney Injury (AKI) develops hyperkalemia. Why does this primarily occur?
O Decreased aldosterone secretion
O Increased potassium intake
O Reduced renal excretion of potassium
O Potassium shifting into cells
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In this question, we're asked why a client with Acute Kidney Injury, or A K I, develops hyperkalemia. Let's break down the concepts of A K I and potassium balance to find the answer.
Understanding AKI and Hyperkalemia
First, let's look at the terms given. A K I refers to a sudden decrease in kidney function. Hyperkalemia is a condition where the potassium level in the blood becomes too high.
The kidneys play a crucial role in maintaining electrolyte balance by filtering potassium from the blood and excreting it in the urine. Let's visualize this process simple.
In Acute Kidney Injury, the kidney's ability to filter blood, known as the Glomerular Filtration Rate, drops significantly. This means it cannot move potassium into the urine as it normally would.
Now let's evaluate the answer choices. Option one suggests decreased aldosterone. While aldosterone helps excrete potassium, A K I is primarily defined by the failure of the kidney's filtration machinery itself.
Evaluating Options
1. Decreased aldosterone secretion (Secondary effect)
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