AKI and Hyperkalemia

MedicinePathophysiologyMedium

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A client with Acute Kidney Injury (AKI) develops hyperkalemia. Why does this primarily occur?

O Decreased aldosterone secretion

O Increased potassium intake

O Reduced renal excretion of potassium

O Potassium shifting into cells

Animated Video Solution

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Step by Step Written Solution

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Step 1

In this question, we're asked why a client with Acute Kidney Injury, or A K I, develops hyperkalemia. Let's break down the concepts of A K I and potassium balance to find the answer.

Understanding AKI and Hyperkalemia

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Step 2

First, let's look at the terms given. A K I refers to a sudden decrease in kidney function. Hyperkalemia is a condition where the potassium level in the blood becomes too high.

$$AKI = \text{Acute Kidney Injury}$$
$$\text{Hyperkalemia} = \text{High Blood Potassium (K}^+)$$
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Step 3

The kidneys play a crucial role in maintaining electrolyte balance by filtering potassium from the blood and excreting it in the urine. Let's visualize this process simple.

Blood K+KidneyUrine Output
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Step 4

In Acute Kidney Injury, the kidney's ability to filter blood, known as the Glomerular Filtration Rate, drops significantly. This means it cannot move potassium into the urine as it normally would.

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Step 5

Now let's evaluate the answer choices. Option one suggests decreased aldosterone. While aldosterone helps excrete potassium, A K I is primarily defined by the failure of the kidney's filtration machinery itself.

Evaluating Options

1. Decreased aldosterone secretion (Secondary effect)

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About This Question

Subject
Medicine
Topic
Pathophysiology
Difficulty
Medium
Question Type
Multiple Choice

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